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Vetalog (triamcinolone acetonide 0.5 mg) Dailymed

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Approved by FDA under NADA # 137-694

For oral use in dogs and cats only.


Federal law restricts this drug to use by or on the order of a licensed veterinarian.


Triamcinolone acetonide is a highly potent synthetic glucocorticoid and anti-inflammatory agent. 1 Its advantage over the older corticoids lies in its ability to achieve equal anti-inflammatory effect with a lower dose. 2 3 4 Triamcinolone has very weak sodium-retaining effects and is probably the least electrolyte-retaining compound of the corticosteroid group. 2 4 Triamcinolone has a plasma half-life of approximately 300 minutes and is classified as an intermediate acting glucocorticoid, whereas the acetonide salt has a longer duration of action and a higher lipid-water distribution coefficient. 1 2 3 4


Triamcinolone acetonide is a highly potent glucocorticoid effective in the treatment of inflammation and related disorders in dogs and cats. It is indicated in the management and treatment of acute arthritis and allergic and dermatologic disorders.

Dosage And Administration:

The keystone of satisfactory therapeutic management with triamcinolone acetonide, as with other steroids, is individualization of dosage in reference to the severity of the disease, the anticipated duration of steroid therapy and the animal’s threshold or tolerance for steroid excess. The prime objective of steroid therapy should be to achieve a satisfactory degree of control with a minimum effective dose.

The initial suppressive dose level of 0.5 - 1.0 mg per 10 pounds of body weight daily should be administered until a satisfactory clinical response is obtained, a period not to exceed 14 days. If a satisfactory response is not obtained in 14 days, re-evaluation of the case to confirm the original diagnosis should be made. As soon as a satisfactory clinical response is obtained, the daily dose should be reduced gradually, either to termination of treatment in the case of acute conditions (e.g., seasonal asthma, dermatitis, acute ocular inflammations) or to the minimal effective maintenance dose level in the case of chronic conditions (e.g., rheumatoid arthritis). Symptoms of adrenal insufficiency following withdrawal may persist for several days, weeks or years. Some cases have resulted in death. To minimize the adverse effects from withdrawal or reduction in dosage, cautiously decrease dosage in a gradual manner. In dogs, dosing in the morning may also be beneficial in minimizing effects because nocturnal pituitary/adrenal activity will be less inhibited. In chronic conditions, and in rheumatoid arthritis especially, it is important that the reduction in dosage from initial to maintenance dose levels be accomplished slowly. The maintenance dose level should be adjusted from time to time as required by fluctuation in the activity of the disease and the animal’s general status. Maintenance dosage levels of 0.125-0.25 mg per 10 pounds of body weight daily are recommended. Accumulated experience has shown that the long-term benefits to be gained from continued steroid maintenance are probably greater the lower the maintenance dose level. In rheumatoid arthritis in particular, maintenance steroid therapy should be at the lowest possible level.

Important: In the therapeutic management of animals with chronic diseases, such as rheumatoid arthritis, triamcinolone should be regarded as a highly valuable adjunct, to be used in conjunction with but not as a replacement for standard therapeutic measures.

Recommended Dosage Schedule:




Daily Dosage


Daily Dosage

5 lbs

0.25 to 0.5 mg

0.0625 to 0.125 mg

10 lbs

0.5 to 1.0 mg

0.125 to 0.25 mg

20 lbs

1.0 to 2.0 mg

0.25 to 0.50 mg

30 lbs

1.5 to 3.0 mg

0.375 to 0.75 mg

60 lbs

3.0 to 6.0 mg

0.75 to 1.50 mg


Do not use in viral infections. Except for emergency therapy, do not use in animals with tuberculosis, chronic nephritis, cushingoid syndrome and peptic ulcers. Existence of congestive heart failure, diabetes and osteoporosis are relative contraindications.


Not for human use. Clinical and experimental data have demonstrated that corticosteroids administered orally or parenterally to animals may induce the first stage of parturition when administered during the last trimester of pregnancy and may precipitate premature parturition followed by dystocia, fetal death, retained placenta and metritis.

Additionally, corticosteroids administered to dogs, rabbits and rodents during pregnancy have produced cleft palate. Other congenital anomalies including deformed forelegs, phocomelia and anasarca have been reported in the offspring of dogs which received corticosteroids during pregnancy.


Because of its inhibitory effect on fibroplasia, triamcinolone may mask the signs of infection and enhance dissemination of the infecting organism. Hence, all animals receiving triamcinolone should be watched for evidence of intercurrent infection. Should infection occur, it must be brought under control by use of appropriate antibacterial measures or administration of triamcinolone should be discontinued.

Because this anti-inflammatory steroid manifests little sodium-retaining activity, the usual early sign of cortisone or hydrocortisone overdosage (i.e., increase in body weight due to fluid retention) is not a reliable index of overdosage. Hence, recommended dosage levels should not be exceeded, and all animals receiving triamcinolone acetonide should be under close medical supervision.

Use of corticosteroids may result in the inhibition of endogenous steroid production which sometimes persists for weeks following drug withdrawal. In patients presently receiving or recently withdrawn from corticosteroid treatments, administration of a rapid acting corticosteroid before, during and after an unusually stressful situation is recommended.

Single or multiple doses of 1 mg/kg of a corticosteroid induced hepatopathy in the dog and rabbit in a study by Rogers and Ruebner in 1977. 4 The condition was determined in the dog by a biopsy of the liver and was accompanied by elevated serum glutamic-pyruvic transaminase, SAP and SGGT (in some dogs) and increased bromsulphalein retention.

Adverse Reactions:

As with the use of any corticosteroid, side effects and metabolic alterations can be anticipated when treatment is intensive or prolonged. In animals with diabetes mellitus, use of triamcinolone acetonide may be associated with an increase in the insulin requirement. Negative nitrogen balance may occur, particularly in animals that require protracted maintenance therapy.3 4 5

Polydipsia or polyuria may occur with high dosage or frequent administration. The likelihood of their occurrence may be minimized by giving as brief a course of corticosteroid therapy as possible, and by waiting for the reappearance of symptoms before repeating therapy. If polydipsia or polyuria should occur, therapy should then be resumed at a lower dosage level.

Other adverse reactions that have occurred with the use of corticosteroids are SAP and SGPT enzyme elevations, weight loss, anorexia, vomiting and diarrhea (occasionally bloody). Anaphylactoid reactions have occasionally been seen following administration. Cushing’s syndrome in dogs has been reported in association with prolonged or repeated steroid therapy.

To report suspected adverse events, for technical assistance, or to obtain a copy of the Safety Data Sheet, contact Boehringer Ingelheim Animal Health USA Inc. at 1-888-637-4251.

For additional information about adverse drug experience reporting for animal drugs, contact FDA by telephone at 1-888-FDA-VETS or online at www.fda.gov/reportanimalae.

Clinical Pharmacology:

Glucocorticoids exert a regulatory influence on lymphocytes, erythrocytes and eosinophils of the blood and on the structure and function of lymphoid tissues.1 4 5 A primary feature of the glucocorticoids is their anti-inflammatory activity with minimum sodium and water retention, which is often associated with the mineralocorticoids. 1 2 3 4 5 6 Glucocorticoids not only inhibit the early phases of the inflammatory process (edema, fibrin deposition, capillary dilatation, migration of leukocytes into the inflamed area and phagocytic activity) but also the later manifestations (capillary proliferation, fibroblast proliferation and deposition of collagen). 3 4 6 The exact mechanism is not known, but the glucocorticoids obviously suppress normal tissue response to injury and alleviate symptoms from many conditions.2

As with other adrenal steroids, triamcinolone acetonide has been found useful in alleviating the pain and lameness associated with acute localized arthritic conditions and generalized arthritic conditions. Glucocorticoids have been used successfully to treat traumatic arthritis, osteoarthritis and generalized arthritic conditions in dogs. Remission of musculoskeletal conditions may be permanent, or symptoms may recur, depending on the cause and extent of structural degeneration. 1 2 3 4 5 Glucocorticoids also relieve pruritus and inflammation of allergic dermatitis, acute moist dermatitis, dry eczema, urticaria, bronchial asthma, pollen sensitivities and otitis externa in dogs and allergic dermatitis and moist and dry eczema in cats. Symptoms may be expected to recur if the cause of the allergic reaction is still present, in which case retreatment may be indicated. In treating acute hypersensitivity reactions, such as anaphylactic shock, appropriate treatment such as intravenous prednisolone sodium succinate should be used.1 2 3 4 5

In dogs and cats moribund from overwhelmingly severe infections for which antibacterial therapy is available (e.g., critical pneumonia, pyometritis), a glucocorticoid may be lifesaving, acting to inhibit the inflammatory reaction, which itself may be lethal; preventing vascular collapse and preserving the integrity of the blood vessels; modifying the animal’s reaction to drugs; and preventing or reducing the exudative reaction which often complicates certain infections. As supportive therapy, it improves the general attitude of the animal being treated. All necessary procedures for the establishment of a bacterial diagnosis should be carried out whenever possible before institution of therapy. Corticosteroid therapy in the presence of infection should be administered for the shortest possible time compatible with maintenance of an adequate response, and antibacterial therapy should be continued for at least three days after the hormone has been withdrawn. Combined hormone and antibacterial therapy does not obviate the need for indicated surgical treatment.1 2 3 4 5 6


Store at 20° to 25°C (68° to 77°F), excursions permitted between 15° and 30°C (between 59º and 86ºF).

How Supplied:

VETALOG Tablets are available in two strengths, 0.5 and 1.5 mg.

NDC 0010-6881-01 - 0.5 mg - 1000 tablets

NDC 0010-6882-01 - 1.5 mg - 500 tablets


1. Osol, A., ed., 1980. Remington's Pharmaceutical Sciences, 16th Edition. Mack Publishing Company, Easton, PA. 898-901, 911.

2. Martindale, The Extra Pharmacopoeia, 27th Edition, 1977. The Pharmaceutical Press, London, England. 389-396, 432-433.

3. Gilman, A.G., L.S. Goodman and A. Gilman, eds., 1980. The Pharmacological Basis of Therapeutics, 6th Edition. Macmillan Publishing Co., Inc., New York, NY. 1470-1496.

4. Booth, N.H. and L.E. McDonald, eds., 1982. Veterinary Pharmacology and Therapeutics, 5th Edition. The Iowa State University Press, Ames, IA. 553-570.

5. DiPalma, J.R., ed., 1976. Basic Pharmacology in Medicine. McGraw-Hill, Inc., St. Louis, MO. 328-337.

6. Kirk, R.W., ed., 1980. Current Veterinary Therapy VII, Small Animal Practice. W.B. Saunders Company, Philadelphia, PA. 497-500, 992-994.

VETALOG is a registered trademark of Boehringer Ingelheim Animal Health USA Inc.



Revised 08/2019

Principal Display Panel Container Label, 0.5 Mg, 1000 Tablets

NDC 0010-6881-01


(triamcinolone acetonide)


Caution: Federal law restricts this drug to use by or on the order of a licensed veterinarian.

Net Contents: 1000 Tablets

Approved by FDA under NADA # 137-694

0.5 mg

Principal Display Panel Container Label, 1.5 Mg, 500 Tablets

NDC 0010-6882-01


(triamcinolone acetonide)


Caution: Federal law restricts this drug to use by or on the order of a licensed veterinarian.

Net Contents: 500 Tablets

Approved by FDA under NADA # 137-694

1.5 mg


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